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Paper Details

Reciprocal inhibition of PIN1 and APC/C<sub>CDH1</sub> controls timely G1/S transition and creates therapeutic vulnerability.
Res Sq
0
2023
APC/C, CDH1, CDK4/6, CDKs, Cyclin-dependent kinases, E3 ligase, E3 ubiquitin ligase, G1, PIN1, PIN1 ubiquitin ligases, anaphase-promoting complex, anti-cancer, cancer, proline, proline isomerase, triple-negative breast cancer
Author NameAffiliation
Yansheng LiuYale Cancer Biology Institute
Yansheng LiuYale University School of Medicine
Felipe BataliniHarvard T.H. Chan School of Public Health
Nathanael S GrayChem-H and Stanford Cancer Institute, Stanford University
Nathanael S GrayChem-H and Stanford Cancer Institute, Stanford University
Gerburg M WulfDepartment of Medicine and Cancer Research Institute, Beth Israel Deaconess Medical Center, Harvard Medical School
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