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Paper Details

UV-induced histone H2AX phosphorylation and DNA damage related proteins accumulate and persist in nucleotide excision repair-deficient XP-B cells.
41
2011
-, ATM, DDR proteins, DNA, DNA DSB, DSB, FANCD2, H2AX, Mdm2, NBS1, NER deficient XP-B cells, Rad51, Wip1, Wip1 phosphatase, XP, XP-, XP-B, XP-B cells, XPB DNA repair helicase, deficient rodent cells, dipyrimidine, global genome repair-deficient rodent cells, histone H2AX, human, normal cells, normal human fibroblasts, nucleotide, nucleotide excision repair-deficient XP-B cells, p-ATM, p-NBS1, p21, p53, rodent

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