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Paper Details

N-terminal acetylation shields proteins from degradation and promotes age-dependent motility and longevity.
Nat Commun
6
2023
Drosophila, Drosophila NatC, KCMF1, N, N-terminal acetyltransferase, NatC, UBR, UBR4, UbcE2M, acetyltransferase NatC, developmental muscle defects, human, human cells, loss of motility, male sterility, methionine, muscle, ubiquitin, ubiquitin ligases
Author NameAffiliation
Michael CostanzoUniversity of Toronto
Amy Hin Yan TongUniversity of Toronto
Brenda J AndrewsUniversity of Toronto
Brenda J AndrewsUniversity of Toronto
Jason MoffatUniversity of Toronto
Jason MoffatUniversity of Toronto
Jason MoffatThe Hospital for Sick Children
Chad L MyersUniversity of Minnesota-Twin Cities
Chad L MyersUniversity of Minnesota-Twin Cities
Chad L MyersUniversity of Minnesota-Twin Cities
Chad L MyersUniversity of Minnesota-Twin Cities
Charles BooneUniversity of Toronto
Charles BooneUniversity of Toronto
Charles Boone
Charles BooneUniversity of Toronto
Charles BooneUniversity of Toronto
Charles Boone
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