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Paper Details

ApoC-III ASO promotes tissue LPL activity in the absence of apoE-mediated TRL clearance.
J Lipid Res
41
2019
APOC-III, APOE isoform, APOE isoform genotype, Alb-Cre +, ApoC, ApoC-III, Apoe -/-, CVD, Hypertriglyceridemia, LDL receptor, LDL receptor-related protein 1, LPL, Ndst1, Ndst1 f/f, SDC1, TG, TRL, TRLs, VLDL, apoC, apoC-III, apoE, mice, syndecan-1, triglyceride, volanesorsen
Author NameAffiliation
Max W ChangDepartments of Cellular and Molecular Medicine,University of California
Courtney R GreenUniversity of California
Ronald M KraussChildren's Hospital Oakland Research Institute Oakland
Christian M MetalloUniversity of California
Christopher BennerDepartments of Cellular and Molecular Medicine,University of California
Christopher BennerDepartments of Cellular and Molecular Medicine,University of California
Joseph L WitztumUniversity of California
Sotirios TsimikasUniversity of California
Philip L S M GordtsUniversity of California
Philip L S M GordtsUniversity of California
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