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Paper Details

KMT2C mediates the estrogen dependence of breast cancer through regulation of ERα enhancer function.
Oncogene
84
2018
ER, ER enhancer, ER enhancers, ER- breast cells, ER-positive breast cancer, Estrogen, Estrogen receptor alpha, FOXA1, H3K27ac, H3K4, H3K4 methyltransferase, H3K4me1, KMT2C, KMT2C-depleted cells, breast cancer, cancer, cancer cell types, estrogen, mammary-derived carcinomas, trans-acting elements, tumor
Author NameAffiliation
Amit U Sinha
Francisco VegaWashington University School of Medicine
Ronald C HendricksonMemorial Sloan Kettering Cancer Center
James J HsiehWashington University School of Medicine
James J HsiehWashington University School of Medicine
Michael F BergerMemorial Sloan Kettering Cancer Center
Nikolaus SchultzMemorial Sloan Kettering Cancer Center
Nikolaus SchultzMemorial Sloan Kettering Cancer Center
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