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Paper Details

Genome-wide CRISPR-Cas9 Screens Reveal Loss of Redundancy between PKMYT1 and WEE1 in Glioblastoma Stem-like Cells.
Cell Rep
117
2015
CDK1, CRISPR, Cas9, GBM, GSC, GSCs, Glioblastoma, Glioblastoma Stem-like Cells, Myt1, NSCs, PKMYT1, WEE1, cyclin B, glioblastoma, human, human neural stem/progenitors, patient, patient-derived GBM stem-like cells, wee1-like kinase
Author NameAffiliation
Eunjee LeeIcahn Institute of Genomics and Multiscale Biology, Icahn School of Medicine at Mount Sinai
Eunjee LeeIcahn Institute of Genomics and Multiscale Biology, Icahn School of Medicine at Mount Sinai
Traver HartUniversity of Toronto and Donnelly Centre, Canada Canadian Institute for Advanced Research
Qing ZhangFred Hutchinson Cancer Research Center
Qing ZhangFred Hutchinson Cancer Research Center
Bruce J AronowCincinnati Children's Hospital Medical Center
Bruce J AronowCincinnati Children's Hospital Medical Center
Christopher L PlaisierInstitute for Systems Biology
Nitin S BaligaInstitute for Systems Biology
Nitin S BaligaInstitute for Systems Biology
Jason MoffatUniversity of Toronto and Donnelly Centre, Canada Canadian Institute for Advanced Research
Lin QiTexas Children's Cancer Center, Baylor College of Medicine
Xiao-Nan LiTexas Children's Cancer Center, Baylor College of Medicine
Do-Hyun NamInstitute for Refractory Cancer Research, Samsung Medical Center
Do-Hyun NamInstitute for Refractory Cancer Research, Samsung Medical Center
Jeongwu LeeLerner Research Institute, Cleveland Clinic
Jun ZhuIcahn Institute of Genomics and Multiscale Biology, Icahn School of Medicine at Mount Sinai
Jeffrey J DelrowFred Hutchinson Cancer Research Center
James M OlsonClinical Research Division, Fred Hutchinson Cancer Research Center
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