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Paper Details

Genome-wide association studies suggest that APOL1-environment interactions more likely trigger kidney disease in African Americans with nondiabetic nephropathy than strong APOL1-second gene interactions.
Kidney Int
49
2018
APOL1, APOL1 G1/G2 renal-risk variants, APOL1 risk, APOL1 risk variants, APOL1-associated nephropathy, ESRD, HIV, HIV infection, LN, apolipoprotein L1 gene, apolipoprotein L1 gene (APOL1) renal risk variants, end-stage renal disease, interferon, kidney disease, lupus nephritis, nephropathy, nondiabetic ESRD, nondiabetic nephropathy, novel loci, second gene, single nucleotide polymorphisms, systemic lupus erythematosus
Author NameAffiliation
Carl D LangefeldDepartment of Biostatistical Sciences Wake Forest School of Medicine, USA Center for Public Health Genomics Wake Forest School of Medicine
Carl D LangefeldDepartment of Biostatistical Sciences Wake Forest School of Medicine, USA Center for Public Health Genomics Wake Forest School of Medicine
Maggie C Y NgDepartment of Biochemistry Wake Forest School of Medicine, USA Center for Diabetes Research Wake Forest School of Medicine
Jasmin DiversDepartment of Biostatistical Sciences Wake Forest School of Medicine, USA Center for Public Health Genomics Wake Forest School of Medicine
Jasmin DiversDepartment of Biostatistical Sciences Wake Forest School of Medicine, USA Center for Public Health Genomics Wake Forest School of Medicine
Pamela J HicksDepartment of Biochemistry Wake Forest School of Medicine, USA Center for Diabetes Research Wake Forest School of Medicine
Donald W BowdenDepartment of Biochemistry Wake Forest School of Medicine, USA Center for Diabetes Research Wake Forest School of Medicine
Nicholette D PalmerDepartment of Biochemistry Wake Forest School of Medicine, USA Center for Diabetes Research Wake Forest School of Medicine
Barry I FreedmanCenter for Public Health Genomics Wake Forest School of Medicine, USA Center for Diabetes Research Wake Forest School of Medicine, Section on Nephrology Wake Forest School of Medicine
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