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Paper Details

Dysregulation of mitochondrial bioenergetics and quality control by HIV-1 Tat in cardiomyocytes.
Journal of Cellular Physiology
21
2018
ATP, Ca2+, Cardiovascular disease, HIV, HIV gene, HIV-1 Tat, HIV-encoded Tat protein, HIV-infected cells, HIV-positive, LC3 II, NRVCs, SQSTM1, Tat, cardiac disease, cardiomyocyte, cardiomyocytes, neonatal rat ventricular cardiomyocytes, oxygen, p62, patients, primary cell culture model, rat, ubiquitin, uninfected cells
Author NameAffiliation
Farzaneh G TahrirCenter for Neurovirology, Lewis Katz School of Medicine at Temple University
Farzaneh G TahrirCollege of Engineering, Temple University
Santhanam ShanmughapriyaCenter for Translational Medicine, Lewis Katz School of Medicine at Temple University
Santhanam ShanmughapriyaLewis Katz School of Medicine at Temple University
Taha Mohseni AhooyiCenter for Neurovirology, Lewis Katz School of Medicine at Temple University
Tijana KnezevicCenter for Neurovirology, Lewis Katz School of Medicine at Temple University
Manish K GuptaCenter for Neurovirology, Lewis Katz School of Medicine at Temple University
Christopher D KontosDuke University Medical Center
Joseph M McClungBrody School of Medicine
Muniswamy MadeshCenter for Translational Medicine, Lewis Katz School of Medicine at Temple University
Muniswamy MadeshLewis Katz School of Medicine at Temple University
Jennifer GordonCenter for Neurovirology, Lewis Katz School of Medicine at Temple University
Arthur M FeldmanLewis Katz School of Medicine at Temple University
Arthur M FeldmanCardiovascular Research Center, Lewis Katz School of Medicine at Temple University
Joseph Y CheungCenter for Translational Medicine, Lewis Katz School of Medicine at Temple University
Joseph Y CheungLewis Katz School of Medicine at Temple University
Kamel KhaliliCenter for Neurovirology, Lewis Katz School of Medicine at Temple University
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