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Paper Details

Telomerase variant A279T induces telomere dysfunction and inhibits non-canonical telomerase activity in esophageal carcinomas.
PLoS One
14
2014
-catenin, A1062T, A279T, A279T variant, BRG-1, EsC, EsC cells, Li, Li Fraumeni, Li Fraumeni fibroblasts, MEFs, TERC, TERC del, TERC del 341-360, TERT, TERT variants, Telomerase variant, Wnt, Zeocin, cancer, cancer cells, cancers, chronic inflammatory disorders, esophageal cancer, esophageal cancer cells, esophageal cancers, esophageal carcinomas, hematologic malignancies, human, mouse, non, patients, premalignant esophageal lesions, promoter, telomerase, telomerase RNA component, telomerase reverse transcriptase, telomere, wtTERT
Author NameAffiliation
Yuwei ZhangThoracic and GI Oncology Branch National Cancer Institute
Yuwei ZhangThoracic and GI Oncology Branch National Cancer Institute
Mahadev RaoThoracic and GI Oncology Branch National Cancer Institute
Mahadev RaoThoracic and GI Oncology Branch National Cancer Institute
Julie A HongThoracic and GI Oncology Branch National Cancer Institute
Julie A HongThoracic and GI Oncology Branch National Cancer Institute
Danny WangsaNational Cancer Institute
Danny WangsaNational Cancer Institute
Sandra BurkettNational Cancer Institute
Sandra BurkettNational Cancer Institute
Mary ZhangThoracic and GI Oncology Branch National Cancer Institute
Mary ZhangThoracic and GI Oncology Branch National Cancer Institute
Sichuan XiThoracic and GI Oncology Branch National Cancer Institute
Sichuan XiThoracic and GI Oncology Branch National Cancer Institute
Nasser K AltorkiWeill Cornell Medical Center
Thomas RiedNational Cancer Institute
Thomas RiedNational Cancer Institute
Neal S Youngand Blood Institute
David S SchrumpThoracic and GI Oncology Branch National Cancer Institute
David S SchrumpThoracic and GI Oncology Branch National Cancer Institute
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