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Paper Details

Genome-wide increase in histone H2A ubiquitylation in a mouse model of Huntington's disease.
J Huntingtons Dis
10
2013
HD, Huntington's disease, R6/2 transgenic, TG, TG striatum, binding sites, chromatin, downregulated genes, genome-wide, genomic promoter, histone, histone H2A, monoubiquitylated histone H2A, mouse, neurodegenerative disorder, promoters, striatal neurons, transcribed genes, transcript, uH2A, uH2A at genes
Animals, Brain, Chromatin Immunoprecipitation, Disease Models, Animal, Gene Silencing, Histones, Huntington Disease, Mice, Oligonucleotide Array Sequence Analysis, Reverse Transcriptase Polymerase Chain Reaction, Transcription, Genetic, Transcriptome, Ubiquitination, Ubiquitins
Author NameAffiliation
Karen N McFarlandMass General Institute for Neurodegenerative Disease, Massachusetts General Hospital
Sudeshna DasMass General Institute for Neurodegenerative Disease, Massachusetts General Hospital
Sudeshna DasMass General Institute for Neurodegenerative Disease, Massachusetts General Hospital
Tingting SunMass General Institute for Neurodegenerative Disease, Massachusetts General Hospital
Dmitriy LeyferMass General Institute for Neurodegenerative Disease, Massachusetts General Hospital
Dmitriy LeyferMass General Institute for Neurodegenerative Disease, Massachusetts General Hospital
Mee-Ohk KimMass General Institute for Neurodegenerative Disease, Massachusetts General Hospital
Eva XiaMass General Institute for Neurodegenerative Disease, Massachusetts General Hospital
Gavin R SangreyMass General Institute for Neurodegenerative Disease, Massachusetts General Hospital
Alexandre KuhnBrain Mind Institute, Ecole Polytechnique Federale de Lausanne
Ruth Luthi-CarterBrain Mind Institute, Ecole Polytechnique Federale de Lausanne, College of Medicine, University of Leicester
Timothy Clark (CM4AI)Mass General Institute for Neurodegenerative Disease, Massachusetts General Hospital
Tim ClarkMass General Institute for Neurodegenerative Disease, Massachusetts General Hospital
Ghazaleh Sadri-VakiliMass General Institute for Neurodegenerative Disease, Massachusetts General Hospital
Jang-Ho J ChaMass General Institute for Neurodegenerative Disease, Massachusetts General Hospital
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