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Paper Details

EBV Negative Lymphoma and Autoimmune Lymphoproliferative Syndrome Like Phenotype Extend the Clinical Spectrum of Primary Immunodeficiency Caused by STK4 Deficiency.
Front Immunol
26
2018
-lymphoma, ALPS, Autoimmune, Autoimmune Lymphoproliferative Syndrome, B cell lymphoma, CD4, CD4 lymphopenia, CD4+, CD4+ T cells, DNT cell, EBV, EBV Negative Lymphoma, Epstein Barr Virus, Fas, Fas receptor, G, Hodgkin lymphoma, IVIG, Lymphoma, Lymphoproliferative Syndrome, P2, PID, Patient, Primary Immunodeficiency, STK4, STK4 Deficiency, STK4 deficiency, STK4 deficient, STK4 mutation, Serine, Serine/threonine kinase 4, Serine/threonine kinase 4 (STK4) deficiency, Vitamin B12, autosomal recessive genetic condition, c, c.1103, hematolytic anemia, hepatosplenomegaly, hypergammaglobulinemia, immunoglobulin, lymphadenopathy, lymphoma, lymphopenia, lymphoproliferation, non-malignant lymphadenopathy, otitis, patient, patients, primary immunodeficiency, stem cell, threonine, tumor
Author NameAffiliation
Arndt BorkhardtHematology and Clinical Immunology, University Children's Hospital, Heinrich-Heine-University
Arndt BorkhardtHematology and Clinical Immunology, University Children's Hospital, Heinrich-Heine-University
Ute FischerHematology and Clinical Immunology, University Children's Hospital, Heinrich-Heine-University
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