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Paper Details

HLA-C variants associated with amino acid substitutions in the peptide binding groove influence susceptibility to Kawasaki disease.
Hum Immunol
5
2019
A, Ala, Arg, Asp, CTL, HLA, HLA Class I alleles, HLA-B genes, HLA-C, HLA-C peptide binding groove, HLA-C variants, HLA-C*04:01, HLA-C*07:02, HLA-C*12, HLA-C*12:02, KD, Kawasaki disease, Phe, SNVs, Ser, amino acid, children, cytotoxic T cells, pediatric vasculitis, risk A, risk allele, rs2254556, rs6906846, single nucleotide variants, vasculitis
Author NameAffiliation
Chisato ShimizuUniversity California San Diego
Jihoon KimUniversity California San Diego
Victoria J WrightImperial College London
Long T HoangGenome Institute of Singapore
Adriana H TremouletUniversity California San Diego, Rady Childrens' Hospital San Diego
Martin L HibberdGenome Institute of Singapore
Atsushi TakahashiRIKEN Center for Integrative Medical Sciences, Research Institute
Michiaki KuboRIKEN Center for Integrative Medical Sciences
Kaoru ItoRIKEN Center for Integrative Medical Sciences
Toshihiro TanakaTokyo Medical and Dental University Graduate School of Medical and Dental Sciences
Lachlan J M CoinInstitute for Molecular Bioscience, University of Queensland
Lachlan J M CoinInstitute for Molecular Bioscience, University of Queensland
Jane C BurnsUniversity California San Diego, Rady Childrens' Hospital San Diego
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