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Paper Details

Tau induces blood vessel abnormalities and angiogenesis-related gene expression in P301L transgenic mice and human Alzheimer's disease.
Proc Natl Acad Sci U S A
190
2018
Alzheimer, Alzheimer's disease, CD31, CD31-positive endothelial, CD31-positive endothelial cells, P301L, P301L transgenic, Plau, Serpine1, Tau, Vegfa, angiogenesis, angiogenesis-related genes, blood, brain endothelial cell, cortical atrophy, dementia, human, human RNA datasets, mice, neurodegeneration, neurons, normal capillaries, patients, tau, vascular abnormalities
Aging, Alzheimer Disease, Amyloid beta-Peptides, Angiogenesis Inducing Agents, Animals, Brain, Cells, Cultured, Cerebrovascular Circulation, Humans, Mice, Mice, Transgenic, Neurons, tau Proteins
Author NameAffiliation
Rachel E BennettMassGeneral Institute for Neurodegenerative Disease, Massachusetts General Hospital, Harvard Medical School
Ashley B RobbinsMassGeneral Institute for Neurodegenerative Disease, Massachusetts General Hospital, Harvard Medical School
Miwei HuMassGeneral Institute for Neurodegenerative Disease, Massachusetts General Hospital, Harvard Medical School
Xinrui CaoHarvard T.H. Chan School of Public Health
Rebecca A BetenskyHarvard T.H. Chan School of Public Health
Timothy Clark (CM4AI)MassGeneral Institute for Neurodegenerative Disease, Massachusetts General Hospital, Harvard Medical School
Tim ClarkMassGeneral Institute for Neurodegenerative Disease, Massachusetts General Hospital, Harvard Medical School
Sudeshna DasMassGeneral Institute for Neurodegenerative Disease, Massachusetts General Hospital, Harvard Medical School
Sudeshna DasMassGeneral Institute for Neurodegenerative Disease, Massachusetts General Hospital, Harvard Medical School
Bradley T HymanMassGeneral Institute for Neurodegenerative Disease, Massachusetts General Hospital, Harvard Medical School
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Datasets

Release 1.2