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Paper Details

Modulation of type I interferon responses potently inhibits SARS-CoV-2 replication and inflammation in rhesus macaques.
Sci Immunol
6
2023
-, CD163, CD163 MRC1 inflammatory macrophages, Calu-3 cells, CoV-2, CoV-2-infected RMs, IFN, IFN-2, IFN-I, IFN-modulator, IFN-stimulated genes, IFNmod, ISGs, MRC1, RMs, SARS, SARS-CoV, SARS-CoV-, SARS-CoV-2, Siglec-1, Type I interferons, antiviral IFN-stimulated genes, circulating monocytes, coronavirus disease, endogenous IFN, infected, inflammatory cells, rhesus macaques, severe, severe acute respiratory syndrome coronavirus, type I interferon
Author NameAffiliation
Jennifer Tisoncik-GoCenter for Innate Immunity and Immune Disease, University of Washington School of Medicine
David E GordonEmory University
Sudhir Pai KasturiEmory National Primate Research Center, Emory University
Sudhir Pai KasturiEmory University
Michael GaleCenter for Innate Immunity and Immune Disease, University of Washington School of Medicine
Jacob D EstesVaccine and Gene Therapy Institute, Oregon Health & Science University
Jacob D EstesOregon National Primate Research Center, Oregon Health & Science University
Jacob D EstesAarhus University
Jacob D EstesCollege of Science, RMIT University
Gideon SchreiberWeizmann Institute of Science
Steven E BosingerEmory National Primate Research Center, Emory University
Steven E BosingerEmory NPRC Genomics Core Emory National Primate Research Center, Emory University
Steven E BosingerEmory University
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