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Paper Details

SARS-CoV-2 hijacks p38β/MAPK11 to promote virus replication.
mBio
3
2023
COVID, COVID-19, MAPK, MAPK11, SARS, SARS-, SARS-CoV, SARS-CoV-2, SARS-CoV-2-infected cells, Severe, Severe acute respiratory syndrome coronavirus, cell lines, coronavirus disease 2019, human, human cells, infected cells, mRNA, mitogen-activated protein kinase, p38, p38 MAPK, p38 substrates
Author NameAffiliation
Benjamin E Nilsson-PayantIcahn School of Medicine at Mount Sinai
Andrew P KurlandIcahn School of Medicine at Mount Sinai
Andrew P KurlandGlobal Health and Emerging Pathogens Institute, Icahn School of Medicine at Mount Sinai
Chengjin YeTexas Biomedical Research Institute
Yaron TomerMeyer Cancer Center
Yaron TomerEnglander Institute for Precision Medicine, Institute for Computational Biomedicine Weill Cornell Medicine
Yaron TomerColumbia University Vagelos College of Physicians and Surgeons
Oded DanzigerIcahn School of Medicine at Mount Sinai
Brad R RosenbergIcahn School of Medicine at Mount Sinai
Lewis C CantleyMeyer Cancer Center
Lewis C CantleyDana-Farber Cancer Institute, Harvard Medical School
Lewis C CantleyHarvard Medical School
Luis Martinez-SobridoTexas Biomedical Research Institute
Benjamin R tenOeverNew York University Langone Health
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Release 1.2