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Paper Details

Type 2 Diabetes Variants Disrupt Function of SLC16A11 through Two Distinct Mechanisms.
Cell
98
2017
Author NameAffiliation
Josep M MercaderBroad Institute of Harvard and MIT, USA Diabetes Unit and Center for Genomic Medicine, Massachusetts General Hospital
Daniel G TenenBroad Institute of Harvard and MIT, Beth Israel Deaconess Medical Center
Daniel G TenenBroad Institute of Harvard and MIT, Beth Israel Deaconess Medical Center
Marcin von GrotthussBroad Institute of Harvard and MIT
Pierre FontanillasBroad Institute of Harvard and MIT
Clary B ClishBroad Institute of Harvard and MIT, USA Broad Institute of Harvard and MIT
Clary B ClishBroad Institute of Harvard and MIT, USA Broad Institute of Harvard and MIT
Steven A CarrBroad Institute of Harvard and MIT
Steven A CarrBroad Institute of Harvard and MIT
Bridget K WagnerBroad Institute of Harvard and MIT
Maggie C Y NgCenter for Genomics and Personalized Medicine Research, Center for Diabetes Research, Wake Forest School of Medicine
Federico Centeno-CruzInstituto Nacional de Medicina Genomica
Carlos ZerrweckThe Obesity Clinic at Hospital General Tlahuac
Lorena OrozcoInstituto Nacional de Medicina Genomica
David AltshulerBroad Institute of Harvard and MIT, Harvard Medical School, Massachusetts General Hospital
David AltshulerBroad Institute of Harvard and MIT, Harvard Medical School, Massachusetts General Hospital
Stuart L SchreiberBroad Institute of Harvard and MIT
Stuart L SchreiberBroad Institute of Harvard and MIT
Jose C FlorezBroad Institute of Harvard and MIT, USA Diabetes Unit and Center for Genomic Medicine, Massachusetts General Hospital, Harvard Medical School
Suzanne B R JacobsBroad Institute of Harvard and MIT, USA Diabetes Unit and Center for Genomic Medicine, Massachusetts General Hospital
Eric S LanderBroad Institute of Harvard and MIT, Harvard Medical School
Eric S LanderBroad Institute of Harvard and MIT, Harvard Medical School
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