Skip to Main Content

Paper Details

Tumor edge-to-core transition promotes malignancy in primary-to-recurrent glioblastoma progression in a PLAGL1/CD109-mediated mechanism.
Neurooncol Adv
12
2020
C, CD109, CD109high, CD133, CD133low, E, Glioblastoma, PLAGL1, PROM1, Patients, TIC, TICs, edge-TICs, glioblastoma, matched primary and recurrent samples, mouse, patient, patient-, patients, primary and recurrent samples, tumor, tumor-initiating cells
Author NameAffiliation
Hee Jin ChoResearch Institute for Future Medicine, Samsung Medical Center
Hee Jin ChoInstitute for Refractory Cancer Research, Samsung Medical Center, Sungkyunkwan University School of Medicine
Do-Hyun NamResearch Institute for Future Medicine, Samsung Medical Center
Do-Hyun NamInstitute for Refractory Cancer Research, Samsung Medical Center, Sungkyunkwan University School of Medicine
Do-Hyun NamSamsung Medical Center, Sungkyunkwan University School of Medicine
Do-Hyun NamResearch Institute for Future Medicine, Samsung Medical Center
Do-Hyun NamInstitute for Refractory Cancer Research, Samsung Medical Center, Sungkyunkwan University School of Medicine
Do-Hyun NamSamsung Medical Center, Sungkyunkwan University School of Medicine
  • 1 - 8

Datasets

Release 1.2