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Paper Details

Lyl1-deficiency promotes inflammatory responses and increases mycobacterial burden in response to <i>Mycobacterium tuberculosis</i> infection in mice.
Front Immunol
0
2022
-deficient macrophages, CXCL1, CXCL5, IL-1, Lyl1, Lyl1-deficiency, Lyl1-deficient macrophages, Lymphoblastic leukemia 1, MAP kinase, NF, NO, hematopoietic stem cell, leukemia, mature innate cells, mice, mouse, neutrophil, neutrophilic inflammation, nitric oxide, pro
Author NameAffiliation
Harukazu SuzukiLaboratory for. Cellular Function Conversion Technology RIKEN Center for Integrative Medical Sciences
Harukazu SuzukiLaboratory for. Cellular Function Conversion Technology RIKEN Center for Integrative Medical Sciences
Frank BrombacherInternational Centre for Genetic Engineering and Biotechnology
Frank BrombacherUniversity of Cape Town, Institute of Infectious Diseases and Molecular Medicine (IDM), Division of Immunology and South African Medical Research Council (SAMRC) Immunology of Infectious Diseases
Frank BrombacherInstitute of Infectious Disease and Molecular Medicine (IDM), University of Cape Town
Reto GulerInternational Centre for Genetic Engineering and Biotechnology
Reto GulerUniversity of Cape Town, Institute of Infectious Diseases and Molecular Medicine (IDM), Division of Immunology and South African Medical Research Council (SAMRC) Immunology of Infectious Diseases
Reto GulerInstitute of Infectious Disease and Molecular Medicine (IDM), University of Cape Town
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