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Paper Details

Mutagenic mechanisms of cancer-associated DNA polymerase ϵ alleles.
Nucleic Acids Res
9
2021
-4 cells, DNA polymerase alleles, M644G, P286R, P301R, POL2, POL2 ultra-mutator, Pol, Pol allele, Pol exonuclease domain alleles, Saccharomyces cerevisiae, alleles, amino acid, cancer, cancer-associated DNA polymerase alleles, colorectal cancers, exonuclease domain mutant cells, human, human DNA polymerase, mismatched primer end, mutator phenotype, pol2, pol2-4, pol2-4 cells, pol2-M644G, pol2-P301R, yeast
Author NameAffiliation
David J AdamsThe Wellcome Sanger Institute
Stephen P JacksonThe Wellcome/Cancer Research UK Gurdon Institute and Department of Biochemistry, University of Cambridge
Stephen P JacksonThe Wellcome/Cancer Research UK Gurdon Institute and Department of Biochemistry, University of Cambridge
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