Skip to Main Content

Paper Details

Recurrent patterns of DNA copy number alterations in tumors reflect metabolic selection pressures.
Mol Syst Biol
48
2017
18F-fluorodeoxy-glucose, CNA, CNA amplification regions, CNAs, DNA copy number, FDG, cancer, endogenous loci, enolase enzymes, experimental systems, glycolytic genes, hexokinase, human, human tumors, immortalization system, metabolic genes, oncogenes, p53, patient, tumor, tumor suppressor genes, tumors
Author NameAffiliation
Nicholas A GrahamCrump Institute for Molecular Imaging, David Geffen School of Medicine, University of California los angeles
Nicholas A GrahamDavid Geffen School of Medicine, University of California los angeles
Nicholas A GrahamUniversity of Southern California
Johanna Ten HoeveCrump Institute for Molecular Imaging, David Geffen School of Medicine, University of California los angeles
Johanna Ten HoeveDavid Geffen School of Medicine, University of California los angeles
Nikolaus SchultzMarie-Josee and Henry R. Kravis Center for Molecular Oncology, Memorial Sloan Kettering Cancer Center
Nikolaus SchultzMemorial Sloan Kettering Cancer Center
Nikolaus SchultzMarie-Josee and Henry R. Kravis Center for Molecular Oncology, Memorial Sloan Kettering Cancer Center
Nikolaus SchultzMemorial Sloan Kettering Cancer Center
Heather R ChristofkDavid Geffen School of Medicine, University of California los angeles
Heather R ChristofkJonsson Comprehensive Cancer Center, David Geffen School of Medicine, University of California los angeles
Heather R Christofkuniversity of california los angeles Metabolomics Center, David Geffen School of Medicine, University of California
  • 1 - 12

Datasets

Release 1.2