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Paper Details

The TP53 Apoptotic Network Is a Primary Mediator of Resistance to BCL2 Inhibition in AML Cells.
Cancer Discov
205
2019
AML, AML Cells, AML cell lines, BAX, BCL2, BCL2 family members, BCL2L1, Beat AML, Cas9, PMAIP1, TP53, TP53 knockout cells, TRK, acute myeloid leukemia, genes, patient, venetoclax
Animals, Antineoplastic Agents, Antineoplastic Combined Chemotherapy Protocols, Apoptosis, Bridged Bicyclo Compounds, Heterocyclic, Cell Line, Tumor, Drug Resistance, Neoplasm, Humans, Leukemia, Myeloid, Acute, Mice, Mice, Inbred NOD, Mice, SCID, Mitochondria, Proto-Oncogene Proteins c-bcl-2, Sulfonamides, Tumor Suppressor Protein p53, Xenograft Model Antitumor Assays
Author NameAffiliation
Tamilla NechiporukOregon Health and Science University
Tamilla NechiporukKnight Cancer Institute, Oregon Health and Science University
Stephen E KurtzOregon Health and Science University
Stephen E KurtzKnight Cancer Institute, Oregon Health and Science University
Olga NikolovaKnight Cancer Institute, Oregon Health and Science University
Olga NikolovaOregon Health and Science University
Courtney L JonesUniversity of Colorado Denver
Angelo D'AlessandroUniversity of Colorado Denver
Angelo D'AlessandroUniversity of Colorado Denver
Rachel Culp-HillUniversity of Colorado Denver
Amanda d'AlmeidaOregon Health and Science University
Amanda d'AlmeidaKnight Cancer Institute, Oregon Health and Science University
Sunil K JoshiOregon Health and Science University
Sunil K JoshiKnight Cancer Institute, Oregon Health and Science University
Mara RosenbergOregon Health and Science University
Mara RosenbergKnight Cancer Institute, Oregon Health and Science University
Mara RosenbergOregon Health and Science University
Mara RosenbergKnight Cancer Institute, Oregon Health and Science University
Cristina E TognonOregon Health and Science University
Cristina E TognonKnight Cancer Institute, Oregon Health and Science University
Cristina E TognonHoward Hughes Medical Institute, Oregon Health and Science University
Alexey V DanilovOregon Health and Science University
Alexey V DanilovKnight Cancer Institute, Oregon Health and Science University
Brian J DrukerOregon Health and Science University
Brian J DrukerKnight Cancer Institute, Oregon Health and Science University
Brian J DrukerHoward Hughes Medical Institute, Oregon Health and Science University
Bill H ChangKnight Cancer Institute, Oregon Health and Science University
Bill H ChangOregon Health and Science University
Shannon K McWeeneyKnight Cancer Institute, Oregon Health and Science University
Shannon K McWeeneyDepartment of Medical Informatics and Clinical Epidemiology, Oregon Health and Science University
Jeffrey W TynerOregon Health and Science University
Jeffrey W TynerKnight Cancer Institute, Oregon Health and Science University
Jeffrey W TynerKnight Cancer Institute, Oregon Health and Science University
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