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Paper Details

Combined transcriptome and proteome profiling of the pancreatic β-cell response to palmitate unveils key pathways of β-cell lipotoxicity.
BMC Genomics
27
2020
-, -1E cells, -cells, 85 upregulated, BACH1, ER, FOXO1, GLP-1 receptor, LXR, PKA, PKA anchoring proteins, PPAR, Palmitate, RNA, SLC7, SLC7 family amino-acid transporters, amino-acid, binding sites, cAMP, cholesterol, diabetes, fatty acid, fatty acids, gene, human, human islets, insulin, insulin-secreting INS, lipotoxicity, palmitate, palmitate-, palmitate-exposed -cells, palmitate-modified genes, pancreatic -cell, protein kinase A, transcription, treated -cells, type 2 diabetes
Author NameAffiliation
Henrik J JohanssonClinical Proteomics Mass Spectrometry, Karolinska Institutet
Janne Lehti??Clinical Proteomics Mass Spectrometry, Karolinska Institutet
Lorella MarselliUniversity of Pisa
Piero MarchettiUniversity of Pisa
Decio L EizirikULB Center for Diabetes Research, Universite Libre de Bruxelles
Miriam CnopULB Center for Diabetes Research, Universite Libre de Bruxelles
Miriam CnopErasmus Hospital, Universite Libre de Bruxelles
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