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Paper Details

A proteasome-resistant fragment of NIK mediates oncogenic NF-κB signaling in schwannomas.
Hum Mol Genet
5
2019
HGF, MET proto-oncogene, MET proto-oncogene receptor tyrosine kinase, Merlin, Merlin-deficient schwannoma, NF-B, NF-B signaling elements, NF2, NF2-encoded protein, NIK, NIK fragment, NIK kinase domain fragment, SC, SCs, Schwann cells, Schwannomas, c-Met, hepatocyte growth factor, human, murine, neurofibromatosis type 2, normal SCs, nuclear factor kappa-light-chain-enhancer of activated B cells (NF-B)-inducing kinase, patients, schwannoma, schwannomas, tumor, tumors, tyrosine
Author NameAffiliation
Anat Stemmer-RachamimovMassachusetts General Hospital
Gary L JohnsonUniversity of North Carolina
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