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Senataxin suppresses the antiviral transcriptional response and controls viral biogenesis.
Nat Immunol
41
2015
ALS4, AOA2, RNA polymerase II, RNAPII, SETX, SETX-deficient cells, Senataxin, West Nile virus, amyotrophic lateral sclerosis, ataxia with oculomotor apraxia, genes, human, human helicase, inborn errors in SETX, influenza A virus, neurodegenerative diseases, neurologic disorders, patients, senataxin
Author NameAffiliation
Alexander RialdiIcahn School of Medicine at Mount Sinai
Jessica Sook Yuin HoInstitute of Molecular and Cell Biology
Natasha MoshkinaIcahn School of Medicine at Mount Sinai
Zuleyma PeraltaIcahn Institute for Genomics and Multiscale Biology, Icahn School of Medicine at Mount Sinai
Zuleyma PeraltaIcahn Institute for Genomics and Multiscale Biology, Icahn School of Medicine at Mount Sinai
Justine C NoelIcahn School of Medicine at Mount Sinai
Camilla MelegariIcahn School of Medicine at Mount Sinai
Sven HeinzThe Salk Institute for Biological Studies
Sven HeinzThe Salk Institute for Biological Studies
Christopher BennerThe Salk Institute for Biological Studies
Christopher BennerThe Salk Institute for Biological Studies
John A T Young
Christopher F BaslerIcahn School of Medicine at Mount Sinai
Ana Fernandez-SesmaIcahn School of Medicine at Mount Sinai
Harm van Bakel1] Icahn Institute for Genomics and Multiscale Biology, Icahn School of Medicine at Mount Sinai
Harm van Bakel1] Icahn Institute for Genomics and Multiscale Biology, Icahn School of Medicine at Mount Sinai
Ivan MarazziIcahn School of Medicine at Mount Sinai, USA. [2] Global Health and Emerging Pathogens Institute
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