Skip to Main Content

Paper Details

Deletion of Ptpn11 (Shp2) in cardiomyocytes causes dilated cardiomyopathy via effects on the extracellular signal-regulated kinase/mitogen-activated protein kinase and RhoA signaling pathways.
Circulation
66
2008
Erk, Erk1/2, Heart failure, MAPK, PTP, PTPs, Ptpn11, RhoA, SH2 domain-containing PTP, Shp2, Shp2-deficient, Shp2-deficient primary cardiomyocytes, cardiac disease, cardiac dysfunction, cardiac hypertrophy, cardiomyocyte, cardiomyocytes, dilated cardiomyopathy, extracellular signal-regulated kinase, mice, mitogen-activated protein kinase, primary cardiomyocytes, protein tyrosine kinases, protein tyrosine phosphatases, tyrosine

Datasets

Release 1.2