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Paper Details

Dilated cardiomyopathy mutant tropomyosin mice develop cardiac dysfunction with significantly decreased fractional shortening and myofilament calcium sensitivity.
Circ Res
76
2007
Ca(2, Ca(2+), DCM, Dilated cardiomyopathy, Glu40Lys, Glu54Lys, TM, TM protein, actin, alpha, alpha-TM, alpha-TM54 mutation, alpha-tropomyosin, beta-myosin heavy chain, brain natriuretic peptide, calcium, cardiac dysfunction, cardiac myofibers, dilated cardiomyopathy, familial hypertrophic cardiomyopathy, heart failure, human, humans, mice, mouse, mutant alpha-TM, mutant tropomyosin, myofilament, ryanodine, ryanodine receptor, sarcomeric thin filament protein, sarcoplasmic reticulum Ca(2+)-ATPase, skeletal actin, striated muscle alpha-tropomyosin, tropomyosin

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