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Paper Details

Commensal bacteria contribute to insulin resistance in aging by activating innate B1a cells.
Sci Transl Med
107
2018
4-1BB receptor, 4-1BBL, 4-1BBL+, 4BL cells, A. muciniphila, Akkermansia muciniphila, B1a cells, CCR2, CCR2+, CCR2+ monocytes, IR, butyrate, dysregulation of the immune, enrofloxacin, humans, hyperglycemia, innate 4-1BBL+ B1a cells, innate B1a cells, insulin, macaques, metabolic disorders, mice, obesity
Author NameAffiliation
Fedor GusevVavilov Institute of General Genetics, Russian Academy of Sciences
Olga D CarlsonLaboratory of Clinical Investigation, National Institute on Aging
Kevin G BeckerNational Institute on Aging
Kevin G BeckerNational Institute on Aging
Luigi FerrucciNational Institute on Aging
Luigi FerrucciNational Institute on Aging
Giorgio TrinchieriNational Cancer Institute
Giorgio TrinchieriNational Cancer Institute
Evgeny I RogaevVavilov Institute of General Genetics, Russian Academy of Sciences
Evgeny I RogaevCenter for Genetics and Genetic Technologies, Lomonosov Moscow State University
Evgeny I RogaevBrudnick Neuropsychiatric Research Institute, University of Massachusetts Medical School
Josephine M EganLaboratory of Clinical Investigation, National Institute on Aging
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