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Paper Details

Flavivirus Antagonism of Type I Interferon Signaling Reveals Prolidase as a Regulator of IFNAR1 Surface Expression.
Cell Host Microbe
93
2015
Human, Human fibroblasts, IFN, IFN-/, IFN-I, IFNAR1, IFNAR2, NS5, PEPD, Prolidase, Type I Interferon, Type I interferon, West Nile virus, cellular dipeptidase, encephalitic flaviviruses, humans, patients, prolidase, prolidase deficiency, tick-borne encephalitis virus
Author NameAffiliation
Shelly J RobertsonNational Institute of Allergy and Infectious Diseases
Kristin L McNallyNational Institute of Allergy and Infectious Diseases
Travis R TaylorCollege of Medicine, University of Toledo Health Science Campus
Richard GreenUniversity of Washington
Daniel L KastnerNational Human Genome Research Institute
Michael G KatzeUniversity of Washington
Michael G KatzeUniversity of Washington
Alexandra F FreemanLaboratory of Clinical Infectious Diseases, National Institute of Allergy and Infectious Diseases
Sonja M BestNational Institute of Allergy and Infectious Diseases
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