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Paper Details

Repression of CTSG, ELANE and PRTN3-mediated histone H3 proteolytic cleavage promotes monocyte-to-macrophage differentiation.
Nat Immunol
28
2021
-, CTSG, Chromatin, ELANE, H3N, NSPs, PRTN3, actively transcribed genes, amino, autoinflammatory disease, cathepsin G, chromatin, genome, histone, histone H3, histone H3 amino terminus, human, human peripheral blood monocytes, immune cell, macrophage, macrophages, monocyte, monocytes, monocytic cell line, monocytic cells, neutrophil elastase, neutrophil serine proteases, patients, permissive chromatin, primary monocytes, proteinase 3, serine, systemic juvenile idiopathic arthritis
Author NameAffiliation
Peggie CheungInstitute for Immunity, Stanford University School of Medicine
Peggie CheungStanford University School of Medicine
Sarah E ChangInstitute for Immunity, Stanford University School of Medicine
Sarah E ChangStanford University School of Medicine
Mai DvorakInstitute for Immunity, Stanford University School of Medicine
Mai DvorakStanford University School of Medicine
Michele DonatoInstitute for Immunity, Stanford University School of Medicine
Michele DonatoCenter for Biomedical Informatics Research, Stanford University School of Medicine
Joshua E Elias
Or GozaniStanford University
Purvesh KhatriInstitute for Immunity, Stanford University School of Medicine
Purvesh KhatriCenter for Biomedical Informatics Research, Stanford University School of Medicine
Purvesh KhatriInstitute for Immunity, Stanford University School of Medicine
Purvesh KhatriCenter for Biomedical Informatics Research, Stanford University School of Medicine
Paul J UtzInstitute for Immunity, Stanford University School of Medicine
Paul J UtzStanford University School of Medicine
Alex J KuoInstitute for Immunity, Stanford University School of Medicine
Alex J KuoStanford University School of Medicine
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