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Paper Details

Genomic analyses of flow-sorted Hodgkin Reed-Sternberg cells reveal complementary mechanisms of immune evasion.
Blood Adv
87
2019
2p/2p15, 6p21.32, 6q23, 9p/9p24, B2M, CCGs, Classical Hodgkin lymphoma, EBV, EBV-, EBV- cHLs, Epstein-Barr virus-positive (EBV+) tumors, GNA13, HRS, HRS cells, JAK, MSI, NF-B, PD-, PD-1, PI3K, STAT, STAT6, TNFAIP3, XPO1, XPO1 mutations, activation-induced cytidine deaminase, apolipoprotein B mRNA, cHL, cHLs, cancer, cancer candidate genes, carcinogen, chromosome 9p/9p24, cytidine, cytosine- phosphate-guanines, flow cytometry, flow-sorted Hodgkin Reed-Sternberg cells, inflammatory/immune cell, major histocompatibility complex class I, malignant Hodgkin Reed-Sternberg (HRS) cells, nuclear factor B, primary mediastinal B-cell lymphomas, programmed cell death protein 1, tumors
Author NameAffiliation
Chip StewartBroad Institute of Harvard and Massachusetts Institute of Technology
Chip StewartBroad Institute of Harvard and Massachusetts Institute of Technology
David WuUniversity of Washington
Jaegil KimBroad Institute of Harvard and Massachusetts Institute of Technology
Atanas KamburovBroad Institute of Harvard and Massachusetts Institute of Technology
Matthew Ducar
Matthew DucarCenter for Cancer Genome Discovery
Aaron R ThornerDana-Farber Cancer Institute
Aaron R ThornerCenter for Cancer Genome Discovery
Gad GetzHarvard Medical School
Gad GetzBroad Institute of Harvard and Massachusetts Institute of Technology
Gad Getz
Gad GetzCenter for Cancer Research, Massachusetts General Hospital
Gad GetzHarvard Medical School
Gad GetzBroad Institute of Harvard and Massachusetts Institute of Technology
Gad Getz
Gad GetzCenter for Cancer Research, Massachusetts General Hospital
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