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Paper Details

Endothelial <i>GNAQ</i> p.R183Q Increases ANGPT2 (Angiopoietin-2) and Drives Formation of Enlarged Blood Vessels.
Arterioscler Thromb Vasc Biol
23
2022
-Weber syndrome, AEB071, ANGPT2, Angiopoietin-2, CM, CONCLUSIONS Gq-R183Q, Capillary malformation, DSCR, DSCR1, Down syndrome, Down syndrome critical region protein, EC, EC-, EC-R183Q, EC-WT, ECs, Endothelial, Endothelial GNAQ, Enlarged, G-protein q subunit, GNAQ, Gq, NF-B, PKC, PLC (, PLC3, R183Q, Sturge, Sturge-Weber syndrome, YM-254890, angiopoietin-2, brain CM, c.548G, c.548G>A, calcineurin, endothelial Gq-R183Q, endothelial cells, human, mRNA, mice, mutant, mutant Gq, mutant versus, normal human endothelial colony forming cells, nuclear factor kappa B, p.R183Q, phospholipase C), proangiogenic, protein kinase C, shRNA, wild-type EC, wild-type GNAQ
Author NameAffiliation
Jonathan PevsnerKennedy Krieger Institute, Johns Hopkins University School of Medicine
Sanda AlexandrescuBoston Children's Hospital and Harvard Medical School
David ZurakowskiBoston Children's Hospital and Harvard Medical School
Mustafa SahinBoston Children's Hospital and Harvard Medical School
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