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Paper Details

Accumulation of Molecular Aberrations Distinctive to Hepatocellular Carcinoma Progression.
Cancer Res
15
2020
-catenin, 16q, 4q, AKT, CDH1, CTNNB1, CTNNB1 mutation, Cancer, CpG, CpG island, CpG islands, HCC, Hepatocellular Carcinoma, MLL protein family, PI3K, RB, RB1, RB1 gene, SNF, SWI, TERT, TP53, WNT, WNT target genes, WNT targets, because -catenin, cadherin, cell-cycle genes, downstream targets, driver gene, hepatocellular carcinoma, p53, target genes
Author NameAffiliation
Shiro FukudaUniversity of Tokyo
Shiro FukudaUniversity of Tokyo
Kyle R CovingtonBaylor College of Medicine's Human Genome Sequencing Center
Chad J CreightonBaylor College of Medicine's Human Genome Sequencing Center
David A WheelerBaylor College of Medicine's Human Genome Sequencing Center
David A WheelerBaylor College of Medicine's Human Genome Sequencing Center
Tatsuhiro ShibataNational Cancer Center Research Institute
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