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Paper Details

IRF1 is critical for the TNF-driven interferon response in rheumatoid fibroblast-like synoviocytes : JAKinibs suppress the interferon response in RA-FLSs.
Experimental and Molecular Medicine
38
2019
-activated FLSs, FLSs, IFN, IFN-regulated genes, IRF1, IRF1-deficient, TNF-stimulated FLSs, IRGs, JAK, JAKinib, JAKinibs, Janus kinase, RA, RA-FLSs, Rheumatoid arthritis, STAT, TNF, TNF transgenic, arthritis, autoimmune disease, baricitinib, fibroblast-like synoviocytes, human, human FLSs, interferon, mouse, rheumatoid fibroblast-like synoviocytes, synovial inflammation, tofacitinib, transcription factor interferon regulatory factor 1
Author NameAffiliation
Thomas PapInstitute of Musculoskeletal Medicine, University Hospital Muenster
Hans P KienerMedical University of Vienna
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Release 1.2