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Paper Details

The genetic architecture of plasma kynurenine includes cardiometabolic disease mechanisms associated with the SH2B3 gene.
Sci Rep
3
2021
/LNK, C-reactive protein, CRP, LNK, SH2B3, SH2B3 gene, SH2B3 missense variant, SH2B3 variant, SH2B3-/-, SNPs, cardiometabolic disease, central obesity, endotoxemia, human, humans, inflammatory gene SH2B3, kynurenine, kynurenine-related SNP, mice, rs3184504, single-nucleotide polymorphisms, tryptophan
Author NameAffiliation
Christian M ShafferVanderbilt University Medical Center
Lea K DavisVanderbilt University Medical Center
Gail P JarvikUniversity of Washington
Gail P JarvikUniversity of Washington
Ian B StanawayHarborview Medical Center Kidney Research Institute, University of Washington
Scott J HebbringCenter for Precision Medicine Research, Marshfield Clinic Research Institute
Muredach P ReillyIrving Institute for Clinical and Translational Research and Division of Cardiology, Columbia University Medical Center
Muredach P ReillyIrving Institute for Clinical and Translational Research and Division of Cardiology, Columbia University Medical Center
Robert E GersztenBeth Israel Deaconess Medical Center
Thomas J WangUniversity of Texas Southwestern Medical Center
Thomas J WangUniversity of Texas Southwestern Medical Center
Jonathan D MosleyVanderbilt University Medical Center
Jonathan D MosleyVanderbilt University Medical Center
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Datasets

Release 1.2