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Paper Details

Revealing the Determinants of Widespread Alternative Splicing Perturbation in Cancer.
Cell Rep
35
2017
Cancer, DrAS, Tumors, cancer, driver variant, immune system-related genes, patients, splicing, splicing isoforms, tumors
Alternative Splicing, Gene Regulatory Networks, Genetic Heterogeneity, Humans, Internet, Mutation, Neoplasms, Phenotype
Author NameAffiliation
Yongsheng LiThe University of Texas MD Anderson Cancer Center, USA College of Bioinformatics Science and Technology and Bio-Pharmaceutical Key Laboratory of Heilongjiang Province, Harbin Medical University
Nidhi SahniThe University of Texas MD Anderson Cancer Center, Baylor College of Medicine
Rita PancsaMedical Research Council Laboratory of Molecular Biology
Daniel J McGrailThe University of Texas MD Anderson Cancer Center
Juan XuCollege of Bioinformatics Science and Technology and Bio-Pharmaceutical Key Laboratory of Heilongjiang Province, Harbin Medical University
Xu HuaThe University of Texas MD Anderson Cancer Center
Jasmin Coulombe-HuntingtonInstitute for Research in Immunology and Cancer, University of Montreal
Michael Ryan
Michael Ryan
Boranai TychhonThe University of Texas MD Anderson Cancer Center
Dhanistha SudhakarThe University of Texas MD Anderson Cancer Center
Limei HuThe University of Texas MD Anderson Cancer Center
Mike TyersInstitute for Research in Immunology and Cancer, University of Montreal
Mike TyersInstitute for Research in Immunology and Cancer, University of Montreal
Xiaoqian JiangUniversity of California at San Diego
Shiaw-Yih LinThe University of Texas MD Anderson Cancer Center
Madan Mohan BabuMedical Research Council Laboratory of Molecular Biology
Madan Mohan BabuMedical Research Council Laboratory of Molecular Biology
Song YiThe University of Texas MD Anderson Cancer Center
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