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Paper Details

Krüppel-like factor 6-mediated loss of BCAA catabolism contributes to kidney injury in mice and humans.
Proc Natl Acad Sci U S A
32
2021
AKI, ATP, BCAA, BCAA catabolic gene, BCAA gene, BCAA genes, BCKDHB, BT2, KLF6, Klf6, Krppel-like factor 6, PT, acute kidney injury, branched-chain amino acid, chronic kidney disease, fibrosis, genes, human, humans, injured cells, kidney fibrosis, kidney injury, kidney proximal tubule (PT) cells, mice, patients, promoter region
Author NameAffiliation
Sian E PiretStony Brook University
Yiqing GuoStony Brook University
Ahmed A AttallahStony Brook University
Sylvia J HorneStony Brook University
Amy ZollmanIndiana University
Daniel OwusuStony Brook University
Justina HeneinStony Brook University
Viktoriya S SidorenkoDepartment of Pharmacological Sciences, Stony Brook University
Monica P ReveloUniversity of Utah
Takashi HatoIndiana University
Avi Ma'ayanDepartment of Pharmacological Sciences, Mount Sinai Center for Bioinformatics, Icahn School of Medicine at Mount Sinai
Avi Ma'ayanDepartment of Pharmacological Sciences, Mount Sinai Center for Bioinformatics, Icahn School of Medicine at Mount Sinai
John Cijiang HeDepartment of Pharmacological Sciences, Mount Sinai Center for Bioinformatics, Icahn School of Medicine at Mount Sinai
John Cijiang HeJames J. Peters Veteran Affairs Medical Center
John Cijiang HeIcahn School of Medicine at Mount Sinai
Sandeep K MallipattuStony Brook University
Sandeep K MallipattuNorthport Veterans Affairs Medical Center
Sandeep K MallipattuStony Brook University
Sandeep K MallipattuNorthport Veterans Affairs Medical Center
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Release 1.2