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Paper Details

Loss of proximal tubular transcription factor Krüppel-like factor 15 exacerbates kidney injury through loss of fatty acid oxidation.
Kidney Int
26
2021
AAI, ACAA2, Acaa2, CPT1A, Cpt1a, FAO, FAO gene, FAO genes, KLF15, Klf15, Krppel-like factor 15, PPAR, PPAR binding sites, PPARA, Ppara, Proximal, Tubulointerstitial, acute kidney injury, aristolochic acid I, fatty acid, fibrosis, human, integrin, kidney injury, mice, mouse, promoter region, proximal, proximal tubule, proximal tubule cells, zinc, zinc-finger transcription factor
Author NameAffiliation
Sian E PiretStony Brook University
Ahmed A AttallahStony Brook University
Xiangchen GuStony Brook University, Yueyang Hospital of Integrated Traditional Chinese and Western Medicine
Yiqing GuoStony Brook University
Nehaben A GujaratiStony Brook University
Justina HeneinStony Brook University
Amy ZollmanIndiana University
Takashi HatoIndiana University
Avi Ma'ayanDepartment of Pharmacological Sciences, Mount Sinai Center for Bioinformatics, Icahn School of Medicine at Mount Sinai
Avi Ma'ayanDepartment of Pharmacological Sciences, Mount Sinai Center for Bioinformatics, Icahn School of Medicine at Mount Sinai
Monica P ReveloUniversity of Utah
Kathleen G DickmanDepartment of Pharmacological Sciences, Stony Brook University
Chung-Hsin ChenNational Taiwan University Hospital
Chia-Tung ShunNational Taiwan University Hospital
Thomas A RosenquistDepartment of Pharmacological Sciences, Stony Brook University
John Cijiang HeDepartment of Pharmacological Sciences, Mount Sinai Center for Bioinformatics, Icahn School of Medicine at Mount Sinai
Sandeep K MallipattuStony Brook University, Northport VA Medical Center
Sandeep K MallipattuStony Brook University, Northport VA Medical Center
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Release 1.2