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Paper Details

Absence of miR-146a in Podocytes Increases Risk of Diabetic Glomerulopathy via Up-regulation of ErbB4 and Notch-1.
J Biol Chem
71
2017
BTBR ob/ob, Diabetic Glomerulopathy, EGFR, ErbB, ErbB kinase, ErbB4, MCP-1, MCP-1 induced protein-1, MCPIP1, MicroRNA, MicroRNA-146a, Notch-1, Podocyte, Podocytes, STZ, T2D, TGF, TGF-1, albuminuria, albuminuric, diabetic, diabetic glomerular injury, diabetic glomerulopathy, diabetic kidney disease, erlotinib, glomerular damage, glomeruli, glomerulopathy, hyperglycemia, miR, miR-146a, miR-146a-/, mice, monocyte chemoattractant protein-1, myeloid cells, pEGFR, pErbB4, patients, podocyte, podocytes, streptozotocin, type 2 diabetes
Author NameAffiliation
Katalin SusztakPerelman School of Medicine, University of Pennsylvania
Matthias KretzlerUniversity of Michigan ann arbor
Matthias KretzlerUniversity of Michigan ann arbor
Markus BitzerUniversity of Michigan ann arbor
Vineet Gupta
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