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Paper Details

Absence of full-length dystrophin impairs normal maturation and contraction of cardiomyocytes derived from human-induced pluripotent stem cells.
Cardiovasc Res
43
2020
-, CMs, Ca2, Ca2+, Cas9, DMD, DMD cells, DMD), Dp427, Dp427 deficiency, Duchenne muscular dystrophy (, Heart failure, MD, actin, c, cardiomyocyte, cardiomyocytes, cardiomyopathy, dystrophin, dystrophin (Dp427) deficiency, dystrophin deficiency, dystrophin gene, exon 50, hiPSC, hiPSC-CMs, human, human cardiomyocytes, human-induced pluripotent stem cells, muscular dystrophy, myofibrils, patient, patients
Author NameAffiliation
Michael R HoopmannInstitute for Systems Biology
Robert L MoritzInstitute for Systems Biology
Robert L MoritzInstitute for Systems Biology
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Release 1.2