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Paper Details

Apobec1 complementation factor overexpression promotes hepatic steatosis, fibrosis, and hepatocellular cancer.
Journal of Clinical Investigation
17
2021
A1CF, A1cf, A1cf+/, A1cf+/Tg, A1cf-/-, ApoB mRNA, Apobec1, Apobec1 complementation factor, Apobec1-/-, Cbr3, Cd36, Cdh1, Cidea, Col1a1, Col4a1, Cxcl14, Gpx3, Gstm3, HCC, Il19, Kif20a, Ly6c, Mcm2, Mcm4, Mcm6, Mmp2, Mogat1, Mogat2, RNA, RNA targets, Sox4, Sox9, Tnf, dysplasia, fibrosis, fructose, hepatic steatosis, hepatocellular cancer, hepatocyte-specific A1cf, human, lipogenic gene, mRNA, mRNAs, mice, nonalcoholic fatty liver disease, polysomal RNA, steatosis
Author NameAffiliation
Joseph H NadeauPacific Northwest Research Institute
Lewis R RobertsMayo Clinic College of Medicine and Science
Nicholas O DavidsonWashington University School of Medicine
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