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Paper Details

ABIN1 Determines Severity of Glomerulonephritis via Activation of Intrinsic Glomerular Inflammation.
Am J Pathol
11
2017
ABIN1, ABIN1[, ABIN1[D472N] cultured podocytes, ABIN1[D472N] podocytes, ABIN1[D485N], ABIN1[D845N], D485N, GN, Glomerular Inflammation, Glomerulonephritis, NF-B, Nephrotoxic serum nephritis, Proteinuria, Tumor, Tumor necrosis factor, WT cells, antibody-mediated nephritis, cultured human-derived podocytes, genes, glomerular cells, glomerular inflammatory, glomerulonephritis, human, human neutrophils, mice, mouse, neutrophil, neutrophils, podocyte, podocytes, primary human neutrophils, proteinuria, systemic autoimmunity, ubiquitin, ubiquitin-binding protein
Author NameAffiliation
Celine C BerthierUniversity of Michigan ann arbor
Patrick M GaffneyOklahoma Medical Research Foundation
Kenneth R McLeishDepartment of Medicine University of Louisville, University of Michigan ann arbor
David W PowellDepartment of Medicine University of Louisville
David W PowellDepartment of Medicine University of Louisville
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