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Paper Details

A tension-mediated glycocalyx-integrin feedback loop promotes mesenchymal-like glioblastoma.
Nat Cell Biol
83
2018
GBM, GBMs, Glioblastoma multiforme, Mesenchymal-like GBMs, brain tumours, integrin, mesenchymal, mesenchymal,, mesenchymal, stem-like, mesenchymal-like glioblastoma, stem, stem-like, stem-like phenotype
Animals, Antineoplastic Agents, Alkylating, Brain Neoplasms, Cell Survival, Feedback, Physiological, Glioblastoma, Glycocalyx, Humans, Integrins, Mesenchymal Stem Cells, Mice, Nude, Neoplastic Stem Cells, Surface Tension, Temozolomide, Tumor Cells, Cultured, Xenograft Model Antitumor Assays
Author NameAffiliation
James M BarnesCenter for Bioengineering and Tissue Regeneration, University of California San Francisco
Shelly KaushikCenter for Bioengineering and Tissue Regeneration, University of California San Francisco
Russell BainerCenter for Bioengineering and Tissue Regeneration, University of California San Francisco
Jason K SaInstitute for Refractory Cancer Research, Samsung Medical Center
Jason K SaResearch Institute for Future Medicine, Samsung Medical Center
Elliot C WoodsDepartment of Chemistry and Howard Hughes Medical Institute, Stanford University
FuiBoon KaiCenter for Bioengineering and Tissue Regeneration, University of California San Francisco
Laralynne PrzybylaCenter for Bioengineering and Tissue Regeneration, University of California San Francisco
Mijeong LeeInstitute for Refractory Cancer Research, Samsung Medical Center
Mijeong LeeSungkyunkwan University
Hye Won LeeInstitute for Refractory Cancer Research, Samsung Medical Center
Hye Won LeeSungkyunkwan University School of Medicine
Jason C TungCenter for Bioengineering and Tissue Regeneration, University of California San Francisco
Ori MallerCenter for Bioengineering and Tissue Regeneration, University of California San Francisco
Alexander S BarrettUniversity of Colorado
Kan V LuHelen Diller Cancer Center, University of California San Francisco
Johnathon N LakinsCenter for Bioengineering and Tissue Regeneration, University of California San Francisco
Kirk C HansenUniversity of Colorado
Kirsten Obernier (CM4AI)Helen Diller Cancer Center, University of California San Francisco
Kirsten Obernier (CM4AI)University of California san francisco
Arturo Alvarez-BuyllaHelen Diller Cancer Center, University of California San Francisco
Arturo Alvarez-BuyllaUniversity of California san francisco
Gabriele BergersHelen Diller Cancer Center, University of California San Francisco
Gabriele BergersVlaams Instituut for Biotechnologie-Center for Cancer Biology and KU Leuven
Joanna J PhillipsHelen Diller Cancer Center, University of California San Francisco
Joanna J PhillipsHelen Diller Cancer Center, University of California San Francisco
Do-Hyun NamInstitute for Refractory Cancer Research, Samsung Medical Center
Do-Hyun NamSamsung Medical Center, Sungkyunkwan University School of Medicine
Do-Hyun NamSungkyunkwan University
Do-Hyun NamInstitute for Refractory Cancer Research, Samsung Medical Center
Do-Hyun NamSungkyunkwan University
Do-Hyun NamSamsung Medical Center, Sungkyunkwan University School of Medicine
Carolyn R BertozziDepartment of Chemistry and Howard Hughes Medical Institute, Stanford University
Valerie M WeaverCenter for Bioengineering and Tissue Regeneration, University of California San Francisco
Valerie M WeaverUniversity of California san francisco
Valerie M WeaverUCSF Comprehensive Cancer Center, Helen Diller Family Cancer Research Center, University of California San Francisco
Valerie M WeaverUniversity of California San Francisco
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