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Paper Details

CD22 blockade restores homeostatic microglial phagocytosis in ageing brains.
Nature
245
2019
B cell receptor, CD22, Cas9, Microglia, RNA, aged microglia, ageing, amyloid, and -synuclein, cognitive decline, mice, microglia, microglial, microglial impairment, neurodegenerative disease, sialic acid
Author NameAffiliation
Madeleine K D ScottStanford University School of Medicine
Madeleine K D ScottStanford University School of Medicine
Madeleine K D ScottInstitute for Immunity, Stanford University School of Medicine
Purvesh KhatriInstitute for Immunity, Stanford University School of Medicine
Purvesh KhatriStanford University School of Medicine
Purvesh KhatriInstitute for Immunity, Stanford University School of Medicine
Purvesh KhatriStanford University School of Medicine
Carolyn R BertozziStanford University School of Medicine
Carolyn R BertozziStanford University
Carolyn R BertozziStanford University
Carolyn R BertozziHoward Hughes Medical Institute, Stanford University
Michael C BassikStanford University
Michael C BassikStanford University School of Medicine
Michael C BassikStanford University
Michael C BassikStanford University School of Medicine
Tony Wyss-CorayStanford University School of Medicine
Tony Wyss-CorayStanford University
Tony Wyss-CorayVeterans Administration Palo Alto Healthcare System
Tony Wyss-CorayPaul F. Glenn Center for the Biology of Aging, Stanford University School of Medicine
Tony Wyss-CorayWu Tsai Neurosciences Institute, Stanford University
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