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Paper Details

LKB1 inactivation modulates chromatin accessibility to drive metastatic progression.
Nat Cell Biol
26
2021
Cas9, LKB1, LKB1-deficient cells, SOX17, cancer, cancer cells, chromatin, driver, early-stage primary tumours, liver kinase B1, lung adenocarcinoma, lung cancer, primary tumours, tumour, tumour suppressor gene
Author NameAffiliation
Jeffrey M GranjaStanford University School of Medicine
Jeffrey M GranjaCenter for Personal and Dynamic Regulomes, Stanford University School of Medicine
M Ryan CorcesCenter for Personal and Dynamic Regulomes, Stanford University School of Medicine
David M FeldserDepartment of Cancer Biology and Abramson Family Cancer Research Institute, Perelman School of Medicine, University of Pennsylvania
Howard Y ChangStanford University School of Medicine
Howard Y ChangCenter for Personal and Dynamic Regulomes, Stanford University School of Medicine
Howard Y ChangStanford University School of Medicine
Michael C BassikStanford University School of Medicine
Michael C BassikStanford University
Michael C BassikStanford University School of Medicine
Michael C BassikStanford University
William J GreenleafStanford University School of Medicine
William J GreenleafCenter for Personal and Dynamic Regulomes, Stanford University School of Medicine
William J GreenleafStanford University School of Medicine
William J GreenleafCenter for Personal and Dynamic Regulomes, Stanford University School of Medicine
Monte M WinslowStanford University School of Medicine
Monte M WinslowStanford University
Monte M WinslowStanford University School of Medicine
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Release 1.2