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Paper Details

Multi-omics: Differential expression of IFN-γ results in distinctive mechanistic features linking chronic inflammation, gut dysbiosis, and autoimmune diseases.
J Autoimmun
18
2020
3' UTR region, ARE, ARE-Del, AU-rich element, FXR, IFN, IFN-, IFN- mRNA, LXRs, PPARs, autoimmune, autoimmune diseases, bile acid, blood, chronic inflammation, fatty acid, gut dysbiosis, immunologic dysfunction, macrophages, mice, murine, nucleotides, tryptophan
Author NameAffiliation
Vishal ThovaraiCenter for Cancer Research, National Cancer Institute-Frederick
Amiran DzutsevCenter for Cancer Research, National Cancer Institute-Frederick
Giorgio TrinchieriCenter for Cancer Research, National Cancer Institute-Frederick
Giorgio TrinchieriCenter for Cancer Research, National Cancer Institute-Frederick
Merrill Eric GershwinAllergy and Clinical Immunology, University of California at Davis
Howard A YoungCenter for Cancer Research, National Cancer Institute-Frederick
Howard A YoungCenter for Cancer Research, National Cancer Institute-Frederick
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