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Paper Details

RARRES2 functions as a tumor suppressor by promoting β-catenin phosphorylation/degradation and inhibiting p38 phosphorylation in adrenocortical carcinoma.
Oncogene
37
2017
-catenin, /, ACC, ACC cell lines, ACC cells, CMKLR1, RARRES2, Retinoic acid, Retinoic acid receptor responder 2, Tumor, Tumor suppressor genes, Wnt, Wnt/-catenin, adrenocortical carcinoma, anticancer immune cells, cancer, chemerin chemokine receptor 1, immune, immunodeficient mouse xenograft models, malignancy, mouse, p38, p38 mitogen-activated protein kinase, promoting, tumor, tumor suppressor gene
Adrenal Cortex Neoplasms, Adrenocortical Carcinoma, Animals, Cell Line, Tumor, Chemokines, Female, Heterografts, Humans, Intercellular Signaling Peptides and Proteins, Male, Mice, Neoplasm Transplantation, Phosphorylation, Proteolysis, Tumor Suppressor Proteins, Wnt Signaling Pathway, beta Catenin, p38 Mitogen-Activated Protein Kinases
Author NameAffiliation
Yi Liu-ChittendenNational Cancer Institute, National Institutes of Health
Yi Liu-ChittendenNational Cancer Institute, National Institutes of Health
Meenu JainNational Cancer Institute, National Institutes of Health
Meenu JainNational Cancer Institute, National Institutes of Health
Kelli GaskinsNational Cancer Institute, National Institutes of Health
Kelli GaskinsNational Cancer Institute, National Institutes of Health
Shubin WangNational Cancer Institute, National Institutes of Health
Shubin WangNational Cancer Institute, National Institutes of Health
Maria J MerinoNational Cancer Institute, National Institutes of Health
Maria J MerinoNational Cancer Institute, National Institutes of Health
Shweta KotianNational Cancer Institute, National Institutes of Health
Shweta KotianNational Cancer Institute, National Institutes of Health
Sudheer Kumar GaraNational Cancer Institute, National Institutes of Health
Sudheer Kumar GaraNational Cancer Institute, National Institutes of Health
Sean DavisCenter for Cancer Research, National Cancer Institute
Susan R DavisCenter for Cancer Research, National Cancer Institute
Lisa ZhangNational Cancer Institute, National Institutes of Health
Lisa ZhangNational Cancer Institute, National Institutes of Health
Electron KebebewNational Cancer Institute, National Institutes of Health
Electron KebebewNational Cancer Institute, National Institutes of Health
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