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Paper Details

SLX4IP Antagonizes Promiscuous BLM Activity during ALT Maintenance.
Mol Cell
57
2019
ALT, ALT cancer, ALT cancer cells, ALT telomeres, ALT-positive osteosarcomas, BLM, BTR complexes, Cancer, Cancer cells, SLX4, SLX4IP, SMX, XPF, osteosarcomas, recombining telomeres, telomerase, telomere, telomeres
Author NameAffiliation
Adam LabadorfBoston University School of Medicine
Simon J BoultonThe Francis Crick Institute
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Release 1.2