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Paper Details

SHIP-1, a target of miR-155, regulates endothelial cell responses in lung fibrosis.
FASEB Journal
21
2020
AKT, BLM, EndoMT, Endothelial, Endothelial SHIP-1, Endothelial cells, Fibrosis, HUVEC, HUVEC cells, Inositol, JAK, MLEC, PI3K, Primary mouse lung endothelial cells, SHIP-1, SHIP-1 gene, SMAD, STAT, STAT3, Src Homology 2-containing Inositol Phosphatase-1, TGF-1, VEC, VEC-Cre-miR-155, VEC-SHIP-1, VEC-miR-155, bleomycin, endothelial cell, endothelial miR, endothelial miR-155, fibrosis, human, human umbilical vein endothelial cells, lung fibrosis, lung inflammation, miR, miR-155, miR-155KO, mice, mouse
Author NameAffiliation
Zhou ZhuSection of Allergy and Clinical Immunology, Yale University School of Medicine
Zhou ZhuBrown University Warren Alpert Medical School
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