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Paper Details

An altered extracellular matrix-integrin interface contributes to Huntington's disease-associated CNS dysfunction in glial and vascular cells.
Hum Mol Genet
3
2023
Astrocytes, CNS dysfunction, Drosophila, HD, Huntington's disease, ITG, ITGs, astrocytes, brain endothelial cells, brain microvascular endothelial cells, glia, glial and vascular cells, human, human iPSC-derived cells, iPSC, iPSC-derived cells, induced pluripotent stem cell, integrin, neurodegeneration, neurovascular unit, patient
Author NameAffiliation
Ryan G LimInstitute for Memory Impairments and Neurological Disorders, University of California Irvine
Mark A Dane
Rebecca Smith-Bindman
Rebecca Smith-Bindman
Kaylyn Devlin
Jie WuUniversity of California Irvine
Malcolm CasaleUniversity of California Irvine
David Kilburn
Laura M Heiser
Laura M HeiserOHSU Knight Cancer Institute
Laura M Heiser
Laura M HeiserOHSU Knight Cancer Institute
James E Korkola
James E KorkolaOHSU Knight Cancer Institute
Juan BotasBaylor College of Medicine
Juan BotasJan and Dan Duncan Neurological Research Institute at Texas Children's Hospital
Juan BotasBaylor College of Medicine
Juan BotasBaylor College of Medicine
Leslie M ThompsonUniversity of California Irvine
Leslie M ThompsonInstitute for Memory Impairments and Neurological Disorders, University of California Irvine
Leslie M ThompsonUniversity of California Irvine
Leslie M ThompsonCenter for the Neurobiology of Learning and Memory, University of California irvine
Leslie M ThompsonSue and Bill Gross Stem Cell Research Center, University of California Irvine
Leslie M ThompsonUniversity of California Irvine
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