Skip to Main Content

Paper Details

EGFR suppresses p53 function by promoting p53 binding to DNA-PKcs: a noncanonical regulatory axis between EGFR and wild-type p53 in glioblastoma.
Neuro Oncol
10
2022
CM-H2DCFDA, Cas9, DNA-PKcs, DNA-dependent protein kinase catalytic subunit, EGFR, EGFR-knockout GSCs, Epidermal growth factor receptor, GBM, GSCs, TP53, glioblastoma, glioblastoma multiforme, glioma, glioma sphere-forming cells, mice, nedisertib, p53, p53 interacting protein, p53 target genes, wild-type p53, wt, wt-p53
Author NameAffiliation
Amanda R WasylishenThe University of Texas MD Anderson Cancer Center
Dimpy KoulThe University of Texas MD Anderson Cancer Center
Wai-Kwan Alfred YungThe University of Texas MD Anderson Cancer Center
Wai-Kwan Alfred YungThe University of Texas MD Anderson Cancer Center
  • 1 - 4

Datasets

Release 1.2